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Search for publications in the Transfection Database with Polyplus transfection reagents or for transfection conditions.

Over 6000 publications, 1000 cell lines and primary cells available.

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Found 1247 results :
Cell Linein vitro
in vivo
Delivered MoleculeReagentResults & Citations
WI-38in vitrosiRNAINTERFERin
Huang B. et al. (2020)

Aging Cell 19, 13129
Inhibition of histone acetyltransferase GCN5 extends lifespan in both yeast and human cell lines
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LNCaP, PC-3in vitroantimiRINTERFERin
Wang, X. et al. (2014)

BMC Cancer 14, 308
Demethylation of the miR-146a promoter by 5-Aza-2′-deoxycytidine correlates with delayed progression of castration-resistant prostate cancer
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HK-2in vitrosiRNAINTERFERin
Iaconis D. et al. (2020)

Human Mol Genet 29, 1018-1029
The HOPS Complex Subunit VPS39 controls ciliogenesis through autophagy
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T24in vitrosiRNAINTERFERin
Wu, M. J. et al. (2012)

Urol Oncol 30, 69-77
Rictor-dependent AKT activation and inhibition of urothelial carcinoma by rapamycin
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HeLain vitroesiRNAINTERFERin
Diaz-Lopez I. et al. (2019)

Elife 8, 48246
An mRNA-binding channel in the ES6S region of the translation 48S-PIC promotes RNA unwinding and scanning
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A549, NCI-H460in vitrosiRNAINTERFERin
Xu, X. et al. (2014)

Carcinogenesis 35, 2457-66
A signaling pathway consisting of miR-551b, catalase and MUC1 contributes to acquired apoptosis resistance and chemoresistance
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MCF7, T-47Din vitrosiRNAINTERFERin
Zhu J. et al. (2020)

Sci Rep 10, 1049
Pifithrin-
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HEK-293T, Mouse peritoneal macrophages, RAW 264.7in vitroDNA, siRNAINTERFERin, jetPEI
Yao, Z. et al. (2014)

J Biol Chem 289, 9372-9
Death domain-associated protein 6 (Daxx) selectively represses IL-6 transcription through histone deacetylase 1 (HDAC1)-mediated histone deacetylation in macrophages
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HT-29, SW620in vitrosiRNAINTERFERin
Stramucci L. et al. (2019)

Cell Death Dis 10, 842
MKK3 sustains cell proliferation and survival through p38DELTA MAPK activation in colorectal cancer
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MCF7in vitroantimiR, siRNAINTERFERin
Zaccara, S. et al. (2014)

Cell Death Differ 21, 1522-34
p53-directed translational control can shape and expand the universe of p53 target genes
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